Abstract

Author(s): Soares de Sousa, Bruno; De Santana Cirilo, Marry Aneyts; Magalhães Melo, Halanna Celina; Neves de Morais, Caroline; Vasconcelos de Melo, Janatar Stella

Introduction: The decline in kidney function causes changes in calcium metabolism, phosphorus, vitamin D and parathyroid hormone. These changes can result in hyperparathyroidism secondary to chronic kidney disease.

Method: This is a case report of descriptive observational.

Discussion: Secondary hyperparathyroidism can cause calcification of tissues, including the myocardium, putting the individual at risk of life. The treatment is indicated is commonly surgery, however, the patient may evolve with hungry bone syndrome characterized by a severe hypocalcemia above. This must be reversed as soon as possible. However, there isn’t in the scientific literature a recommended dose of calcium by day for this type of patient, so this report is intended to describe the therapeutic use and amount of calcium a day that proved effective in reversing the syndrome.

Results: The calcium supplementation, when combined with food supplementation achieved significant improvement, around 83% in the serum calcium level.

Conclusions: Dietary calcium supplementation optimized the elevated serum calcium levels contributing to the medical treatment of the patient. The increase in the incidence of cases like this, brings the scientific community, the need to state a calcium dose \ Day, where it has been effective in our study was about 3 times the RDA for chronic renal failure patients.